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Draining away brain's toxic protein to stop Alzheimer's
08-12-2007 · EurekAlert!Scientists have shown how the body's natural way of ridding the body of the toxic protein amyloid-beta is flawed in people with the disease. Then the team demonstrated an experimental method in mice to fix the process, dramatically reducing the levels of the toxic protein in the brain and halting symptoms.
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Keywords: draining, away, brain, toxic, protein, stop, alzheimer
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- Discovery supports theory of Alzheimer's disease as form of diabetes
09-26-2007 · EurekAlert!
Insulin may be as important for the mind as it is for the body. Recent research has raised the possibility that Alzheimer's memory loss could be due to a novel third form of diabetes. Scientists at Northwestern University have discovered why brain insulin signaling would stop working in Alzheimer's disease. They have shown that a toxic protein found in the brains of individuals with Alzheimer's removes insulin receptors from nerve cells, rendering those neurons insulin-resistant.
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- Copper damages protein that defends against Alzheimer's
11-06-2007 · EurekAlert!
Copper can damage a molecule that escorts out of the brain a substance called amyloid beta that builds up in toxic quantities in the brains of people with Alzheimer's disease. The new findings demonstrate one way in which copper might contribute to the development of the disease, though scientists say much more research needs to be done to clarify what role, if any, copper ultimately plays.
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- What causes Alzheimer's? Researchers tie beta-amyloids to brain dysfunction
06-04-2007 · EurekAlert!
Researchers tied the accumulation of the toxic brain protein beta-amyloid to Alzheimer's disease, according to a study reported at the 54th Annual Meeting of SNM, the world's largest society for molecular imaging and nuclear medicine professionals.
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- UCLA/Toronto researchers unlock key to memory storage in brain
04-19-2007 · EurekAlert!
Scientists know little about how the brain chooses cells to encode and store memories. Now a UCLA-University of Toronto team has discovered that a protein called CREB controls the odds of a neuron participating in memory formation. The April 20 edition of Science reports the findings, which suggest a new approach for preserving memory in people suffering from Alzheimer's or other brain injury. Memories could be guided into healthy cells and away from sick cells in dying regions of the brain.
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- A possible route to preventing Alzheimer's cognitive decline?
09-05-2007 · EurekAlert!
In experiments with mice, researchers have discovered a mechanism by which the toxic brain protein produced in Alzheimer’s disease (AD) could contribute to the cognitive deficits that are its hallmark. They found evidence that the toxic protein, called Aâ peptide, triggers overexcitation of neurons in the brain’s learning centers, inducing compensatory rewiring of brain circuitry in the centers—all of which could cause deterioration of neural function.
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- A novel way found to prevent protein plaques implicated in Alzheimer's
11-18-2007 · EurekAlert!
For unknown reasons a protein called amyloid beta aggregates into toxic plaques in the brain, killing neurons. These plaques are one of the hallmarks of Alzheimer's disease. Now two new animal studies show for the first time that the deadly transformation of amyloid beta into plaques can be prevented through an interaction between amyloid beta and another protein called cystatin C.
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- New imaging technique identifies people-at-risk for Alzheimer's disease
12-20-2006 · EurekAlert!
UCLA researchers used innovative brain scan technology with a new imaging molecule, invented at UCLA, to show that abnormal brain protein deposits that define Alzheimer's disease can be detected in people with mild cognitive impairment, a condition affecting 15-20 million Americans that increases risk for developing Alzheimer's disease. The new imaging technique helped researchers track disease progression over a two-year period and may be helpful in detecting pre-Alzheimer's conditions.
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- Protein inhibitor tangles with Alzheimer's disease
02-15-2007 · EurekAlert!
A hallmark of Alzheimer's disease (AD) is the abnormal accumulation of phosphorylated forms of a protein known as tau. In a new study, Mayo Clinic researchers have now shown in mice that a drug that inhibits the function of the protein Hsp90 (EC102) reduces the amount of phosphorylated tau in the brain. Unlike many drugs, EC102 is able to cross the blood-brain barrier, making it a highly promising therapeutic candidate for AD.
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- Altering brain's lipid metabolism reduces Alzheimer's plaques in mice
01-17-2008 · EurekAlert!
Increasing levels of a protein that helps the brain use cholesterol may slow the development of Alzheimer's disease changes in the brain, according to researchers studying a mouse model of the disease at Washington University School of Medicine in St. Louis.
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08-15-2007 · EurekAlert!
Scientists have discovered a key protein in the toxic brain pathway that leads to fragile X tremor/ataxia syndrome, an inherited neurodegenerative disorder. The finding, in a Drosophila (fly) model of FXTAS, could help unravel the complex mechanisms of FXTAS and lead scientists to develop therapies to target the protein.
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