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Discovery docks with Station today

10-25-2007 · European Space Agency (ESA)

Space Shuttle Discovery is today scheduled to dock with the International Space Station at 14:35 CEST (12:35 UT). The Shuttle astronauts are due to enter the Station two hours later.

Follow the mission live on NASA TV

Keywords: discovery, docks, station, today, dock

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Reiter to start journey home
12-19-2006 · European Space Agency (ESA)
ESA astronaut Thomas Reiter will start his journey back to Earth later today when Space Shuttle Discovery undocks from the International Space Station. Reiter will have spent a total of 166 days living on the orbiting outpost.
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Discovery prepares for docking
12-11-2006 · European Space Agency (ESA)
Nearly two days after launch, Space Shuttle Discovery is preparing to dock with the International Space Station later this evening. The docking is scheduled to take place at 23:05 CET (22:05 UT).
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Discovery docks with Space Station
12-11-2006 · European Space Agency (ESA)
Space Shuttle Discovery has successfully docked with the International Space Station. Docking took place at 23:12 CET (22:12 UT) as the two spacecraft flew over southeast Asia.
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Atlantis approaches Space Station
02-09-2008 · European Space Agency (ESA)
Space Shuttle Atlantis is scheduled to dock with the International Space Station at 18:25 CET (17:25 UT) today. The hatches between the two spacecraft will be opened 70 minutes later. Follow rendezvous and docking on ESA?s Columbus Blog or live on NASA TV.
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Immune system discovery could aid fight against TB
10-19-2006 · EurekAlert!
A key aspect of how the body kicks the immune system into action against tuberculosis is revealed in research published today. The authors, writing in Science, hope that their research could aid the development of novel vaccines and immunotherapies to combat TB, which is responsible for two million deaths each year.
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Scientists call for global push to advance research in synthetic biology
06-25-2007 · EurekAlert!
With research backgrounds ranging from materials engineering to molecular biophysics, seventeen leading scientists issued a statement today announcing that, much as the discovery of DNA and creation of the transistor revolutionized science, there is a new scientific field on the brink of revolutionizing our approach to problems ranging from eco-safe energy to outbreaks of malaria: synthetic biology.
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STS-120: Crew set for first spacewalk
10-26-2007 · European Space Agency (ESA)
Two astronauts will step out of the International Space Station later today to take part in a six and a half hour spacewalk (Extra Vehicular Activity, EVA) to install the Italian-built Node 2 module, also known as Harmony.
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December Geology and GSA Today media highlights
12-01-2006 · EurekAlert!
Topics include: impact of melting glaciers on near-shore ecosystems; discovery of intact egg clusters from a Middle Cambrian oceanic invertebrate; biotic recovery from the End-Permian mass extinction; fossil evidence of unusual animal foraging behavior; dating the destruction of Herod the Great's harbor at Caesarea; El Niño-related landslides along the Big Sur coastline, and evidence of flowing water at Mars' Erebus crater. The GSA Today science article describes dynamics of ice sheet recession in East Antarctica.
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Advance in understanding of blood pressure gene could lead to new treatments
02-04-2007 · EurekAlert!
Research by scientists at UCL (University College London) has clearly demonstrated for the first time the structure and function of a gene crucial to the regulation of blood pressure. The discovery could be important in the search for new treatments for illnesses such as heart disease, the UK's biggest killer. In a paper published online today in Nature Medicine, the team, led by Professor Patrick Vallance and Dr James Leiper, UCL Department of Medicine, reveal the role of the human gene dimethylarginine dimethylaminohydrolase (DDAH), showing that loss of DDAH activity disrupts nitric oxide (NO) production. NO is critical in the regulation of blood pressure, nervous system functions and the immune system. The role of DDAH is to break down modified amino acids (Asymmetric dimethylarginine (ADMA) and monomethyl arginine (L-NMMA)) that are produced by the body and have been shown to inhibit NO synthase. These molecules accumulate in various disease states including diabetes, renal failure and pulmonary and systemic hypertension, and their concentration in plasma (the fluid component of blood) is strongly predicative of cardiovascular disease and death. In a healthy human body, the majority of ADMA is eliminated through active metabolism by DDAH. Scientists have hypothesised that if DDAH function is impaired, NO production is reduced, and that this could be an important feature of increased cardiovascular risk. To examine this pathway in more detail, the researchers deleted the DDAH gene in mice. These mice went on to develop hypertension, or high blood pressure. They also designed specific inhibitors (small molecules) which bind to the active site of human DDAH. These small molecule inhibitors also induced hypertension in mice, confirming the importance of DDAH in the regulation of blood pressure. Dr Leiper, UCL Medicine, said: “These genetic and chemical approaches to disrupt DDAH showed remarkably consistent results, and provide compelling evidence that loss of DDAH function increases the concentration of ADMA and thereby disrupts vascular NO signalling. “There has been considerable scientific interest in this pathway and the role of ADMA as a novel risk factor, but so far there's been little evidence to support the idea that it's a cause of disease, rather than just a marker. Genes and their pathways are crucial to our understanding of cardiovascular disease and a better understanding of DDAH-1 could lead to important new treatments. “It could help us to establish if genetic variation predisposes certain people to these diseases, or whether environmental factors exert some of their effects through modulation of DDAH activity. “Our research also shows that this pathway could be harnessed therapeutically to limit production of NO in certain situations where too much nitric oxide is a bad thing; for example, hypotension and septic shock. These are some of the biggest problems in intensive care medicine and there is a huge unmet need for drug treatments.” The study, which was carried out at UCL's Rayne Institute, was funded by grants from the British Heart Foundation, the Wellcome Trust and the Medical Research Council. Professor Jeremy Pearson, Associate Medical Director of the British Heart Foundation, said: "The unexpected finding in the 1980s that a simple gas, nitric oxide (NO), is made by cells in the blood vessel wall and is a powerful control of blood vessel relaxation led to the award of the Nobel Prize in 1998 to its discoverers. "More recently, there has been increasing evidence that impairment of NO production is likely to be an important factor in the development of heart and circulatory disease, but the mechanisms responsible are not fully understood. "This study suggests for the first time that the loss of the activity of the enzyme DDAH-1 leads to reduced NO production and may cause heart and circulatory disease. These findings are likely to be important in the search for new ways to optimise the health of our blood vessels." ### Notes for Editors 1. For more information, please contact Ruth Metcalfe in the UCL Media Relations Office on tel: +44 (0)20 7679 9739, mobile: +44 (0)7990 675 947, out of hours: +44 (0)7917 271 364, e-mail: r.metcalfe@ucl.ac.uk2. 'Disruption of methylarginine metabolism impairs vascular homeostasis' is published in the February issue of the journal Nature Medicine. Advance online publication is embargoed to 18.00 GMT (13.00 US Eastern) Sunday 4 February 2007. Journalists can obtain copies of the paper by contacting the UCL Media Relations Office.3. The study was funded by the British Heart Foundation, the Wellcome Trust and the Medical Research Council. About UCL Founded in 1826, UCL was the first English university established after Oxford and Cambridge, the first to admit students regardless of race, class, religion or gender, and the first to provide systematic teaching of law, architecture and medicine. In the government's most recent Research Assessment Exercise, 59 UCL departments achieved top ratings of 5* and 5, indicating research quality of international excellence. UCL is the fourth-ranked UK university in the 2006 league table of the top 500 world universities produced by the Shanghai Jiao Tong University. UCL alumni include Mahatma Gandhi (Laws 1889, Indian political and spiritual leader); Jonathan Dimbleby (Philosophy 1969, writer and television presenter); Junichiro Koizumi (Economics 1969, Prime Minister of Japan); Lord Woolf (Laws 1954, Lord Chief Justice of England & Wales); Alexander Graham Bell (Phonetics 1860s, inventor of the telephone), and members of the band Coldplay.
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Launch countdown for Discovery
10-23-2007 · European Space Agency (ESA)
The countdown to launch of Space Shuttle Discovery continues at NASA's Kennedy Space Center, Florida. Discovery is due to launch today at 17:38 CEST (15:38 GMT) on flight STS-120, a mission to install the Italian-built Node 2 module on the ISS.
For the latest updates see NASA's Shuttle page
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