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Keeping at-risk cells from developing cancer
12-10-2007 · EurekAlert!Researchers at Johns Hopkins have discovered that cancers arising from epigenetic changes -- in this case the inappropriate activation of a normally silent gene -- develop by becoming addicted to certain growth factors. Reporting online in next week's Early Edition of the Proceedings of the National Academies of Sciences, the team shows that blocking this "addiction" can greatly prevent cancer growth.
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Keywords: keeping, at-risk, cells, developing, cancer, risk, cell
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- 2 heads are better than 1: 2 dysfunctional DNA repair pathways kill tumor cells
04-12-2007 · EurekAlert!
Individuals who inherit one mutant copy of any one of about 12 genes that make the proteins of the Fanconi Anemia pathway are at increased risk of developing cancer. This occurs when the remaining "good" copy of the gene becomes mutated in a specific cell type. However, hope of a new treatment for these cancers has now been provided by a new study indicating that inhibiting the protein ATM can kill these cancer cells.
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- Fetal cell 'transplant' could be a hidden link between childbirth and reduced risk of breast cancer
10-02-2007 · EurekAlert!
Some benefits of motherhood are intangible, but one has been validated through biostatistical research: women who bear children have a reduced risk of developing breast cancer. In Seattle, researchers at the University of Washington and Fred Hutchinson Cancer Research Center believe they have identified a source of this protective effect: fetal cells "transplanted" to the mother before birth.
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- Long-term cancer risk follows stem cell transplant recipients
11-27-2006 · EurekAlert!
Hematopoietic stem cell transplant (HSCT) recipients face a significant long-term risk for developing a second cancer, particularly if they were older at the time of transplant or received stem cells from a female donor, according to a new study.
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- Therapeutic peptide frees the protein p73 to kill tumor cells
03-08-2007 · EurekAlert!
p53 suppresses tumor development by inducing tumor cell death. However, targeting p53 for the treatment of cancer is confounded by the fact that genetic mutations cause loss or inactivation of p53 in approximately 50 percent of human cancers. Now, a new study indicates that targeting the p53-related protein p73 in mice induces the regression of established tumors of human origin, leading to the suggestion that p73 might be a viable target for developing anticancer therapeutics.
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- Heart failure: Intervention possibilities from imaging programmed cell loss
04-03-2007 · EurekAlert!
Using a nuclear medicine technique and molecular imaging to "see" programmed cell loss -- the body's normal way of getting rid of unneeded or abnormal cells -- may help in early identification of those individuals who are at risk of developing heart failure, say researchers in the April Journal of Nuclear Medicine.
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- JCI table of contents -- May 17, 2007
05-17-2007 · EurekAlert!
This release contains summaries, links to PDFs and contact information for the following newsworthy papers to be published online, May 17, 2007, in the JCI, including: Role of TRPM8 in the development of prostate cancer; Blocking aggrecanase-2 activity prevents cartilage erosion in arthritis; Keeping red blood cells hydrated in sickle cell disease; Enzymes and exercise curb muscle insulin resistance; Endothelial progenitor cells mend vessel damage with a little help from EphB4; and others.
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- Obesity found to be a risk factor for multiple myeloma
07-19-2007 · EurekAlert!
An obese person is more likely than a lean person to develop multiple myeloma, according to researchers from Brigham and Women's Hospital, Harvard Medical School, and Harvard School of Public Health. Their findings indicate that Body Mass Index (BMI) -- a statistical measure that scales weight to height -- provides an indicator for one’s risk of developing multiple myeloma, a cancer of the blood cells that produce antibodies.
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- Neural stem cell study reveals mechanism that may play role in cancer
09-04-2007 · EurekAlert!
In the dynamic world of the developing brain, neural stem cells give rise to neurons deep within the brain’s fluid-filled ventricles. These newborn neurons then migrate along the stem cell fibers up to the neocortex, the seat of higher cognitive functions. Now, scientists have discovered a key mechanism of this migration -- one that may also play an important role in other developmental processes and diseases, including cancer.
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- Growth factors given with chemotherapy associated with increased risk of blood diseases
02-06-2007 · EurekAlert!
Women with breast cancer who receive compounds that stimulate white blood cell production to help their bodies better tolerate chemotherapy are at an increased risk of developing a type of leukemia or a condition called myelodysplastic syndrome, according to a new study in the February 7, Journal of the National Cancer Institute. The authors note that the absolute risk of the conditions is very small, but that risk should still be taken into consideration when making treatment decisions.
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- Promising approach to a more effective sunscreen
12-03-2007 · EurekAlert!
Chronic sun exposure increases the risk of an individual developing skin cancer, because UV light from the sun can cause genetic mutations that enable skin cells to grow in an uncontrolled manner. Hope for a new sunscreen that can prevent and treat UV light-induced skin cancers has been provided by a new study that demonstrates that applying an agent known as CP-31398 to the skin of mice reduced a specific form of skin cancer.
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