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MIT corrects inherited retardation, autism in mice
12-19-2007 · Massachusetts Institute of Technology (MIT)Researchers at MIT's Picower Institute for Learning and Memory have corrected key symptoms of mental retardation and autism in mice. The work indicates that a certain class of drugs could have the same effect in humans.
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Keywords: mit, corrects, inherited, retardation, autism, mice, correct
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Similar news on "MIT corrects inherited retardation, autism in mice":
- MIT researchers reverse symptoms in mice of leading inherited cause of mental retardation
06-25-2007 · EurekAlert!
Researchers at the Picower Institute at MIT have, for the first time, reversed symptoms of mental retardation and autism in mice. The study may lead to new drug therapies for a leading inherited cause of mental retardation (Fragile X Syndrome) and some types of autism.
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- Fragile X retardation syndrome corrected in mice
12-19-2007 · EurekAlert!
Researchers working with mice have significantly alleviated a wide range of abnormalities due to fragile X syndrome by altering only a single gene, countering the effects of the fragile X mutation. They said their achievement offers the potential for treatment of the disorder, the most common form of inherited mental retardation and a leading identified genetic cause of autism. There is currently no treatment or therapy for fragile X syndrome, whose symptoms include mental retardation, epilepsy and abnormal body growth.
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- MIT researchers reverse retardation in mice
06-25-2007 · Massachusetts Institute of Technology (MIT)
Researchers at the Picower Institute for Learning and Memory at MIT have reversed symptoms of mental retardation and autism in mice. The study suggests that inhibiting a certain enzyme could be an effective therapy for countering FXS symptoms in children.
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- Fragile X syndrome -- A stimulating environment restores neuronal function in mice
05-23-2007 · EurekAlert!
A new study of the malfunctioning neuronal machinery of Fragile X syndrome reveals that it can be restored by a stimulating environment. The study found that mice genetically altered to have the same defect as humans with the disorder benefited from an environment with constantly changing toys and access to "play cages." Fragile X syndrome is the most common form of inherited mental retardation, occurring in 1 in 3600 males and 1 in 4000 to 6000 females.
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- Researchers uncover mechanisms of common inherited mental retardation
01-08-2008 · UT Southwestern Medical Center
Researchers at UT Southwestern Medical Center are uncovering how brain cells are affected in Fragile X syndrome, the most common cause of inherited mental retardation and the most common genetic cause of autism.
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- UT Southwestern researchers uncover mechanisms of common inherited mental retardation
01-08-2008 · EurekAlert!
Researchers at UT Southwestern Medical Center are uncovering how brain cells are affected in Fragile X syndrome, the most common cause of inherited mental retardation and the most common genetic cause of autism.
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- Gene research may help explain autistic savants
02-12-2008 · Massachusetts Institute of Technology (MIT)
Mice lacking a certain brain protein learn some tasks better but also forget faster, according to new MIT work that may explain the phenomenon of autistic savants. The work could also result in future treatments for autism and other disorders.
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- MIT corrects sickle-cell anemia in mice
12-07-2007 · Massachusetts Institute of Technology (MIT)
MIT researchers have successfully treated mice with sickle-cell anemia in a process that begins by directly reprogramming the mice's own cells to an embryonic-stem-cell-like state, without the use of eggs.
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- Novel DNA changes linked to autism
03-24-2007 · Science News Online
Genetic alterations that occur in children without being inherited from the parents contribute to certain cases of autism and related developmental disorders.
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- Buildup of damaged DNA in cells drives aging
12-20-2006 · EurekAlert!
A study being published today in the journal Nature found that mice completely lacking a critical gene for repairing damaged DNA grow old rapidly and have physical, genetic and hormonal profiles very similar to mice that grow old naturally. Furthermore, the premature aging symptoms of the mice led to the discovery of a new type of human progeria, a rare inherited disease in which affected individuals age rapidly and die prematurely.
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