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Healthy men who drink moderately have reduced risk of heart attack

For men with healthy lifestyle habits, drinking moderate amounts of alcohol may be associated with a lower risk of heart attack than drinking heavily or not drinking at all, according to a report in the Oct. 23 issue of Archives of Internal Medicine, one of the JAMA/Archives journals.

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1. Healthy diet and lifestyle behaviors associated with decreased risk of heart attack in women
   10-22-2007 · EurekAlert!
   Women who eat a healthy diet, drink moderate amounts of alcohol, are physically active, maintain a healthy weight and do not smoke have a significantly reduced risk of heart attack, according to a report in the Oct. 22 issue of Archives of Internal Medicine, one of the JAMA/Archives journals.
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2. Further evidence that moderate drinking reduces men's heart attack risk
   10-23-2006 · EurekAlert!
   Even as studies have consistently found an association between moderate alcohol consumption and reduced heart attack risk in men, an important question has persisted: What if the men who drank in moderation were the same individuals who maintained good eating habits, didn't smoke, exercised and watched their weight? How would you know that their reduced risk of myocardial infarction wasn't the result of one or more of these other healthy habits?
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3. Men with hypertension who drink moderate amounts of alcohol may have a lower risk of heart attack
   01-01-2007 · EurekAlert!
   Researchers at the Harvard School of Public Health (HSPH), Beth Israel Deaconess Medical Center and Dutch research institute TNO Quality of Life and Wageningen University, the Netherlands, found that, among hypertensive men, moderate alcohol consumption was associated with a decreased risk of fatal and non-fatal heart attack.
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4. Staying active and drinking moderately is the key to a long life
   01-08-2008 · EurekAlert!
   People who drink moderate amounts of alcohol and are physically active have a lower risk of death from heart disease and other causes than people who don't drink at all, according to new research published in the European Heart Journal. People who neither drink alcohol nor exercise have a 30 to 49 percent higher risk of heart disease than those who either drink, exercise or both.
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5. Diesel exhaust associated with higher heart attack, stroke risk in men
   11-06-2007 · EurekAlert!
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6. Anxiety hikes risk of heart attack, death
   05-14-2007 · EurekAlert!
   New research shows that highly anxious patients with heart disease face nearly double the risk of heart attack or death when compared to those with a more serene outlook on life. Patients whose anxiety intensified over time were in greatest peril, while those who started out highly anxious but later found inner calm markedly reduced their risk. The research appears in the May 22, 2007, issue of the Journal of the American College of Cardiology.
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7. UMass Medical School study finds patients diagnosed with coronary heart disease continue poor diets
   01-30-2008 · EurekAlert!
   More than 13 million Americans have survived a heart attack or have been diagnosed with coronary heart disease, the number one cause of death in the United States. In addition to medications, lifestyle changes, such as a healthy diet and exercise, are known to reduce the risk for subsequent cardiac events. Despite this evidence, a high proportion of heart attack survivors do not follow their doctor's advice to adhere to a healthy diet.
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8. What's good for the heart may be good for the prostate
   02-12-2008 · EurekAlert!
   Men who eat a diet low in fat and red meat but high in vegetables and lean protein and who drink alcohol in moderation may not just be doing their hearts a favor. A new study shows that such a heart-healthy diet may also be good for the prostate.
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9. Advance in understanding of blood pressure gene could lead to new treatments
   02-04-2007 · EurekAlert!
   Research by scientists at UCL (University College London) has clearly demonstrated for the first time the structure and function of a gene crucial to the regulation of blood pressure. The discovery could be important in the search for new treatments for illnesses such as heart disease, the UK's biggest killer. In a paper published online today in Nature Medicine, the team, led by Professor Patrick Vallance and Dr James Leiper, UCL Department of Medicine, reveal the role of the human gene dimethylarginine dimethylaminohydrolase (DDAH), showing that loss of DDAH activity disrupts nitric oxide (NO) production. NO is critical in the regulation of blood pressure, nervous system functions and the immune system. The role of DDAH is to break down modified amino acids (Asymmetric dimethylarginine (ADMA) and monomethyl arginine (L-NMMA)) that are produced by the body and have been shown to inhibit NO synthase. These molecules accumulate in various disease states including diabetes, renal failure and pulmonary and systemic hypertension, and their concentration in plasma (the fluid component of blood) is strongly predicative of cardiovascular disease and death. In a healthy human body, the majority of ADMA is eliminated through active metabolism by DDAH. Scientists have hypothesised that if DDAH function is impaired, NO production is reduced, and that this could be an important feature of increased cardiovascular risk. To examine this pathway in more detail, the researchers deleted the DDAH gene in mice. These mice went on to develop hypertension, or high blood pressure. They also designed specific inhibitors (small molecules) which bind to the active site of human DDAH. These small molecule inhibitors also induced hypertension in mice, confirming the importance of DDAH in the regulation of blood pressure. Dr Leiper, UCL Medicine, said: “These genetic and chemical approaches to disrupt DDAH showed remarkably consistent results, and provide compelling evidence that loss of DDAH function increases the concentration of ADMA and thereby disrupts vascular NO signalling. “There has been considerable scientific interest in this pathway and the role of ADMA as a novel risk factor, but so far there's been little evidence to support the idea that it's a cause of disease, rather than just a marker. Genes and their pathways are crucial to our understanding of cardiovascular disease and a better understanding of DDAH-1 could lead to important new treatments. “It could help us to establish if genetic variation predisposes certain people to these diseases, or whether environmental factors exert some of their effects through modulation of DDAH activity. “Our research also shows that this pathway could be harnessed therapeutically to limit production of NO in certain situations where too much nitric oxide is a bad thing; for example, hypotension and septic shock. These are some of the biggest problems in intensive care medicine and there is a huge unmet need for drug treatments.” The study, which was carried out at UCL's Rayne Institute, was funded by grants from the British Heart Foundation, the Wellcome Trust and the Medical Research Council. Professor Jeremy Pearson, Associate Medical Director of the British Heart Foundation, said: "The unexpected finding in the 1980s that a simple gas, nitric oxide (NO), is made by cells in the blood vessel wall and is a powerful control of blood vessel relaxation led to the award of the Nobel Prize in 1998 to its discoverers. "More recently, there has been increasing evidence that impairment of NO production is likely to be an important factor in the development of heart and circulatory disease, but the mechanisms responsible are not fully understood. "This study suggests for the first time that the loss of the activity of the enzyme DDAH-1 leads to reduced NO production and may cause heart and circulatory disease. These findings are likely to be important in the search for new ways to optimise the health of our blood vessels." ### Notes for Editors 1. For more information, please contact Ruth Metcalfe in the UCL Media Relations Office on tel: +44 (0)20 7679 9739, mobile: +44 (0)7990 675 947, out of hours: +44 (0)7917 271 364, e-mail: r.metcalfe@ucl.ac.uk2. 'Disruption of methylarginine metabolism impairs vascular homeostasis' is published in the February issue of the journal Nature Medicine. Advance online publication is embargoed to 18.00 GMT (13.00 US Eastern) Sunday 4 February 2007. Journalists can obtain copies of the paper by contacting the UCL Media Relations Office.3. The study was funded by the British Heart Foundation, the Wellcome Trust and the Medical Research Council. About UCL Founded in 1826, UCL was the first English university established after Oxford and Cambridge, the first to admit students regardless of race, class, religion or gender, and the first to provide systematic teaching of law, architecture and medicine. In the government's most recent Research Assessment Exercise, 59 UCL departments achieved top ratings of 5* and 5, indicating research quality of international excellence. UCL is the fourth-ranked UK university in the 2006 league table of the top 500 world universities produced by the Shanghai Jiao Tong University. UCL alumni include Mahatma Gandhi (Laws 1889, Indian political and spiritual leader); Jonathan Dimbleby (Philosophy 1969, writer and television presenter); Junichiro Koizumi (Economics 1969, Prime Minister of Japan); Lord Woolf (Laws 1954, Lord Chief Justice of England & Wales); Alexander Graham Bell (Phonetics 1860s, inventor of the telephone), and members of the band Coldplay.
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10. Eating whole-grain breakfast cereals may be associated with a lower risk of heart failure for men
    10-22-2007 · EurekAlert!
    Men who consume a higher amount of whole grain breakfast cereals may have a reduced risk of heart failure, according to a report in the Oct. 22 issue of Archives of Internal Medicine, one of the JAMA/Archives journals.
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